Gliomas induce and exploit microglial MT1-MMP expression for tumor expansion

DS Markovic, K Vinnakota… - Proceedings of the …, 2009 - National Acad Sciences
DS Markovic, K Vinnakota, S Chirasani, M Synowitz, H Raguet, K Stock, M Sliwa…
Proceedings of the National Academy of Sciences, 2009National Acad Sciences
Diffuse infiltration of glioma cells into normal brain tissue is considered to be a main reason
for the unfavorable outcomes of patients with malignant gliomas. Invasion of glioma cells
into the brain parenchyma is facilitated by metalloprotease-mediated degradation of the
extracellular matrix. Metalloproteases are released as inactive pro-forms and get activated
upon cleavage by membrane bound metalloproteases. Here, we show that membrane type
1 metalloprotease (MT1-MMP) is up-regulated in glioma-associated microglia, but not in the …
Diffuse infiltration of glioma cells into normal brain tissue is considered to be a main reason for the unfavorable outcomes of patients with malignant gliomas. Invasion of glioma cells into the brain parenchyma is facilitated by metalloprotease-mediated degradation of the extracellular matrix. Metalloproteases are released as inactive pro-forms and get activated upon cleavage by membrane bound metalloproteases. Here, we show that membrane type 1 metalloprotease (MT1-MMP) is up-regulated in glioma-associated microglia, but not in the glioma cells. Overexpression of MT1-MMP is even lethal for glioma cells. Glioma-released factors trigger the expression and activity of MT1-MMP via microglial toll-like receptors and the p38 MAPK pathway, as deletion of the toll-like receptor adapter protein MyD88 or p38 inhibition prevented MT1-MMP expression and activity in cultured microglial cells. Microglial MT1-MMP in turn activates glioma-derived pro-MMP-2 and promotes glioma expansion, as shown in an ex vivo model using MT1-MMP-deficient brain tissue and a microglia depletion paradigm. Finally, MyD88 deficiency or microglia depletion largely attenuated glioma expansion in 2 independent in vivo models.
National Acad Sciences